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 As an example, human SET7/9 has just lately been reported to methylate Lys189 within the general transcription factor TAF10, leading to an increased affinity for RNA polymerase II and transcriptional activation of certain TAF10-dependent genes 37 SET7/9 has additionally been reported to methylate p53, rising the steadiness of this quick lived tumor-suppressor protein 38 These observations recommend that we should not narrowly define the SET-area proteins as histone lysine methyltransferases but instead name them protein lysine methyltransferases.
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